Treatment of multiple infarct dementia, multiple infarct dementia should be treated

multiple infarct dementia treatment considerations? Preventive measures: 1, early detection and avoid risk factors for stroke such as hypertension, diabetes and hyperlipidemia and aggressive treatment of such a high degree of carotid stenosis who may be surgery. Help reduce the incidence of vascular dementia. 2, quit smoking, control alcohol consumption and proper diet. 3, should have a clear genetic background for gene diagnosis and treatment. Health care query Breakline multiple infarct dementia treatments based on Chinese medicine treatment of mental illness in order to calm and soothe the nerves, the principle of stability, together with the drugs have. Role in the treatment of various types of mental illness significantly. Clinically, treatment of most patients, medication 1 week fidget, depression, worry and other symptoms, the patient self-care response. Chinese medicine treatment of mental illness is the most effective formula. Chinese herbal medicines query Breakline multiple infarct dementia Western treatment methods (a) treatment of primary treatment, including treatment of cerebrovascular disease and recovery of brain function in two ways. 1 treatment of hypertension and blood pressure to maintain an appropriate level, can prevent and delay the occurrence of dementia. Some scholars have found, VD patients with hypertension, systolic blood pressure control in the 135 ~ 150mmHg may improve cognitive function, lower than the level of worsening symptoms. (2) to improve cerebral circulation, increase cerebral blood flow, increase oxygen utilization (1) dihydroergotamine bases: elimination of vascular spasm and increase blood flow, improve neuronal function, commonly used double oral ergot 0.5 ~ 1mg, 3 times / d and nicergoline (lysergic bromonicotinate ester). (2) calcium antagonist: increased cerebral blood flow and prevent calcium overload and free radical damage, dihydropyridines such as nimodipine, the treatment of patients with white matter osteoporosis cognitive impairment, a year after stable or improving condition, diphenyl alkyl amines such as flunarizine. (3) nicotinic acid: increased cerebral blood flow and improve memory. (4) medicine: use of PNS (thrombus), puerarin (Bullock forest) and TMP (methylpyrazine), etc., are promoting blood circulation, improve blood viscosity and platelet aggregation. 3 commonly used anti-platelet aggregation aspirin 75 ~ 150mg / d orally, inhibit platelet aggregation, platelet membrane stability, improve cerebral circulation, to prevent thrombosis; ticlopidine (ticlopidine) 250mg / d orally, acting on the cell membrane, a direct impact on platelet adhesion and aggregation, inhibition of platelet between the freeze-dried human fibrinogen (fibrinogen) bridge formation. 4 agent for cerebral metabolism of brain cells to amino acids, phospholipids, and glucose utilization, and enhance the patients response and excitability, enhance memory. (1) pyrrolidone: used piracetam (piracetam) and aniracetam, increase brain adenosine three phosphate (ATP) formation and transfer, increase glucose utilization and protein synthesis, promoting hemispheric information transfer. (2) Meclofenoxate: hormones can play a central role in increasing glucose utilization, and improve the central nervous system excitement of learning and memory. (3) A drug lysergic acid alkali-dihydrotestosterone (DHT ergot alkaloids): Enhanced presynaptic nerve terminals release neurotransmitters, stimulate postsynaptic receptors, improve neurological function and brain cell energy balance. (4) A microphone triazine: If almitrine / Raubasine (all welcome) increase in arterial oxygen pressure and oxygen saturation, increase oxygen, improve microcirculation and cerebral metabolism. (5) others such as brain protein hydrolyzate (Cerebrolysin), citicoline (citicoline), three adenosine monophosphate (ATP), coenzyme A, etc. 5 neuroprotective drugs (1) calcium antagonists: such as nimodipine and flunarizine. (2) excitatory amino acid receptor antagonists: such as magnesium sulfate and MK801. (3) free radical scavengers: like vitamin E, vitamin C and ginkgo biloba and so on. 6 symptomatic treatment (1) patients with depression can be selective 5-HT reuptake inhibitor (SSRI), such as fluoxetine 20mg, 1 times / d, a selective 5 - HT and norepinephrine reuptake inhibitors (SNRI) venlafaxine 25mg, 2 ~ 3 times / d. (2) anxiety disorders can be stable 5mg, 3 times / d, symptoms can be anti-anxiety drug buspirone 30mg / d. (3) patients with insomnia during the day should be in bright light, for the restoration of normal wake cycle to provide a good environment, such as zopiclone available. 7 rehabilitation because of intellectual impairment in vascular dementia often patchy or non-comprehensive, with focal neurological signs, rehabilitation therapy can often receive better treatment. Rehabilitation should be targeted, including daily living skills training, muscle and joint activity training and rehabilitation of speech disorders. Depression and apathy is the spontaneity an important reason for increased dementia, the patient should be made more contacts with the outside world, to participate in certain social activities. Comprehensive treatment by Western medicine, rehabilitation and care to improve patient quality of life, to make some return to society. (B) the prognosis is closely related to the prognosis of cerebrovascular disease, and prognosis of dementia due to the lesion, is also inconsistent range of different, but the general process of cognitive decline, the process was irreversible, progress at different speeds. Medication is essential, and diet could help drug treatment, so multiple infarct dementia of the diet and how diet is like? Drug search Breakline